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Many of the metabolites of phase I are carcinogenic or reactive, so having phase II detox in sync is very important

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LPS also increased lipid peroxidation in rat hearts by reducing GPX4 expression, while the ferroptosis inhibitor Fer-1 alleviated the adverse effects caused by LPS and improved LPS-induced cardiac dysfunction by inhibiting the TLR4/NF-B signaling pathway (197)

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In summary, conserved amino acids are only absent from one half of the acceptor di-peptide binding pocket